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miR-130a Promotes RV Dilation and Ventricular Arrhythmogenesis. (A) Representative <t>echocardiography</t> B-mode images at the mid-ventricular level of control and αMHC-miR130a hearts. Endocardial dimensions of the RV at end diastole are traced (red). (B) Average RV endocardial area for control and αMHC-miR130a hearts (n=5 mice per group; p<0.05) Cardiac Arrhythmogenesis and RV dilation in αMHC-miR130a Mice. (C) Representative surface <t>ECG</t> of control vs. αMHC-miR130a mouse 8 weeks off doxycycline. αMHC-miR130a mice exhibit premature ventricular complexes while age matched control mice maintain a normal sinus rhythm. (D) PVC propensity quantified for both groups (p<0.01; CNTL, n=14; αMHC-miR130a, n=17).
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miR-130a Promotes RV Dilation and Ventricular Arrhythmogenesis. (A) Representative echocardiography B-mode images at the mid-ventricular level of control and αMHC-miR130a hearts. Endocardial dimensions of the RV at end diastole are traced (red). (B) Average RV endocardial area for control and αMHC-miR130a hearts (n=5 mice per group; p<0.05) Cardiac Arrhythmogenesis and RV dilation in αMHC-miR130a Mice. (C) Representative surface ECG of control vs. αMHC-miR130a mouse 8 weeks off doxycycline. αMHC-miR130a mice exhibit premature ventricular complexes while age matched control mice maintain a normal sinus rhythm. (D) PVC propensity quantified for both groups (p<0.01; CNTL, n=14; αMHC-miR130a, n=17).

Journal: MicroRNA (Shariqah, United Arab Emirates)

Article Title: MicroRNA-130a Regulation of Desmocollin 2 in a Novel Model of Arrhythmogenic Cardiomyopathy

doi: 10.2174/2211536605666161109111031

Figure Lengend Snippet: miR-130a Promotes RV Dilation and Ventricular Arrhythmogenesis. (A) Representative echocardiography B-mode images at the mid-ventricular level of control and αMHC-miR130a hearts. Endocardial dimensions of the RV at end diastole are traced (red). (B) Average RV endocardial area for control and αMHC-miR130a hearts (n=5 mice per group; p<0.05) Cardiac Arrhythmogenesis and RV dilation in αMHC-miR130a Mice. (C) Representative surface ECG of control vs. αMHC-miR130a mouse 8 weeks off doxycycline. αMHC-miR130a mice exhibit premature ventricular complexes while age matched control mice maintain a normal sinus rhythm. (D) PVC propensity quantified for both groups (p<0.01; CNTL, n=14; αMHC-miR130a, n=17).

Article Snippet: Electrocardiographic signals were amplified with an ECG Bioamplifier (AD Instruments; Colorado Springs, CO), converted from analog to digital with an ACQ-16 Acquisition Interface and recorded with Ponemah Physiology Platform software (Gould; Valley View, OH).

Techniques: Control